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Journal Impact Factor: 4.4
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Source Normalized Impact per Paper (SNIP): 1.294
SCImago Journal Rank (SJR): 1.518
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Articles
Page 159 of 159
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Citation: Arthritis Research & Therapy 1999 3:66741
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Alternative therapies in rheumatology
Citation: Arthritis Research & Therapy 1999 2:66740 -
How endothelial cells protect themselves from complement by expressing decay-accelerating factor (DAF)
Citation: Arthritis Research & Therapy 1999 3:66739 -
Mutations in the WISP3gene cause PPD
Citation: Arthritis Research & Therapy 1999 3:66738 -
Protection against cartilage and bone destruction by systemic interleukin-4 treatment in established murine type II collagen-induced arthritis
Destruction of cartilage and bone are hallmarks of human rheumatoid arthritis (RA), and controlling these erosive processes is the most challenging objective in the treatment of RA. Systemic interleukin-4 trea...
Citation: Arthritis Research & Therapy 1999 1:81 -
Kinesin-like protein CENP-E is upregulated in rheumatoid synovial fibroblasts
Our aim was to identify specifically expressed genes using RNA arbitrarily primed (RAP)-polymerase chain reaction (PCR) for differential display in patients with rheumatoid arthritis (RA). In RA, amplification...
Citation: Arthritis Research & Therapy 1999 1:71 -
The human search engine [paper reports editorial]
Citation: Arthritis Research & Therapy 1999 1:51 -
New functions for Cox-2 in health and disease: Report of “The Third International Workshop on Cox-2”, Ka’upulehu, Kona, Hawaii, USA, 30 August to 2 September 1999
Citation: Arthritis Research & Therapy 1999 1:45 -
Genetics of rheumatoid arthritis: confronting complexity
The genetic basis for rheumatoid arthritis (RA) is likely to be extremely complex. Even the role of MHC genes remains to be fully defined, and may involve interactive genetic effects. The difficulty of precise...
Citation: Arthritis Research & Therapy 1999 1:37 -
The challenge of early synovitis: multiple pathways to a common clinical syndrome
Citation: Arthritis Research & Therapy 1999 1:31 -
Antigen-specific cytometry
Citation: Arthritis Research & Therapy 1999 1:25 -
Cytokines and direct cell contact in synovitis: relevance to therapeutic intervention
Citation: Arthritis Research & Therapy 1999 1:17 -
Current perspectives on synovitis
Citation: Arthritis Research & Therapy 1999 1:11 -
American College of Rheumatology
Citation: Arthritis Research & Therapy 1999 1:0006 -
Gene therapy in rheumatoid arthritis: how to target joint destruction?
Citation: Arthritis Research & Therapy 1999 1:5 -
Arthritis Research: more than a journal (http://arthritis-research.com)
Citation: Arthritis Research & Therapy 1999 1:1 -
Induction of antiphospholipid antibodies by peptide immunization and proof of their pathogenicity in an animal model
Citation: Arthritis Research & Therapy 1999 3:66737 -
HLA, killer cell inhibitory receptors and autoimmune anemia
Citation: Arthritis Research & Therapy 1999 1:66736 -
The effects of 1α,25-dihydroxyvitamin D3 on matrix metalloproteinase and prostaglandin E2 production by cells of the rheumatoid lesion
The biologically active metabolite of vitamin D3, 1α,25-dihydroxyvitamin D3 [1α,25(OH)2D3], acts through vitamin D receptors, which were found in rheumatoid tissues in the present study. IL-1β-activated rheumatoi...
Citation: Arthritis Research & Therapy 1999 1:63 -
Functional heterogeneity of osteoclasts
Citation: Arthritis Research & Therapy 1999 1:66735 -
New Pathway of NF-?B activation by TNF Described
Citation: Arthritis Research & Therapy 1999 3:66734 -
CD28-deficient MRL/Mpj-lpr/lpr mice
Citation: Arthritis Research & Therapy 1999 3:66733 -
Autoreactivity of T cells selected by a single MHC/peptide ligand
Citation: Arthritis Research & Therapy 1999 3:66732 -
CD30 involvement in self tolerance of T cells
Citation: Arthritis Research & Therapy 1999 3:66731 -
Post-transcriptional regulation of TNF gene expression in vivo
Citation: Arthritis Research & Therapy 1999 3:66730 -
A possible cause of joint destruction in septic arthritis
Citation: Arthritis Research & Therapy 1999 1:3 -
Serum amyloid P component prevents antinuclear autoimmunity
Citation: Arthritis Research & Therapy 1999 3:66729 -
Cathepsin S null mice exhibit relative resistance to collagen-induced arthritis
Citation: Arthritis Research & Therapy 1999 3:66728 -
Galectin 1 suppression of collagen-induced arthritis
Citation: Arthritis Research & Therapy 1999 3:66727 -
The MAPK p38 pathway controls TNF synthesis
Citation: Arthritis Research & Therapy 1999 3:66726 -
Purification and cloning of aggrecanase-1
Citation: Arthritis Research & Therapy 1999 3:66725 -
A T cell receptor transgenic mouse model of inflammatory arthritis
Citation: Arthritis Research & Therapy 1999 3:66724 -
VEGF and bone resorption by osteoclasts
Citation: Arthritis Research & Therapy 1999 1:66723 -
Lessons learned from gene transfer approaches
Recent technological advances allow the transfer of genes to the synovial lining of joints. As well as opening novel opportunities for therapy, these techniques provide valuable new tools for the study of syno...
Citation: Arthritis Research & Therapy 1999 1:21